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Inserm Transfert inserm u.312
Inserm U.312, supplied by Inserm Transfert, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/inserm+u%2E312/inserm+u312/pm09928948-127-8-8
Average 90 stars, based on 1 article reviews
inserm u.312 - by Bioz Stars, 2026-09
90/100 stars

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Article Title: Pro-oxidant role of superoxide dismutase in ultraviolet-A-induced lipid peroxidation in cultured normal human skin fibroblasts.
Article Snippet: 256, 1842189 (1998) FEBS 1998 Pro-oxidant role of superoxide dismutase in ultraviolet-A-induced lipid peroxidation in cultured normal human skin fibroblasts Patrice MORLIÈRE and René SANTUS Laboratoire de Photobiologie, INSERM U.312, Muséum National d’Histoire Naturelle, Paris, France (Received 27 April 1998) 2 EJB 98 0569/6 Exposure of cultured normal human skin fibroblasts to ultraviolet A triggers lipid peroxidation.

Article Title: Tagungskalender
Article Snippet: NCI-EORTC Symposium on New Drugs in Cancer Therapy.. Kontakt: EORTC, New Drug Development Office, Free University Hospital, P.O.. Box 7057, NL-1007 MB Amsterdam.

Article Title: Effects of nitric oxide on the eosinophil survival in vitro. A role for nitrosyl-heme.
Article Snippet: In a previous paper we showed that the nitric oxide (NO) donors azide and hydroxylamine inhibited eosinophil apoptosis.. Azide and hydroxylamine generate a nitrosyl-heme complex ^ due to endogenous catalase activity ^ which activates soluble guanylate cyclase.. In contrast, in the present paper, we show that NO donors (SNAP, SIN-1, S-nitroso-L-cysteine, NOC-18) which spontaneously release NO in physiological solutions did not support the survival of eosinophils and induced apoptosis or necrosis.

Article Title: UVA-induced oxidative damage in fibroblasts cultured in a 3-dimensional collagen matrix.
Article Snippet: The expo.sure of dermal equivalent, i.e., libroblasts cultuied in a 3dimensional collagen tnatrix to realistic doses of UVA (< 200 k.l/nr), results in a lipid peroxidation process as evideticed by the release of thiobarbitutie aeid-reactive substances in the supernatant.. This peioxidative process is shown to be associated with the presence of libroblasts and is inhibited by pr-eincubation with vitamin E, the well-known chain-breaking aritioxidant.. Mor"eover, the UVA inadiation tr iggers cytotoxic effects which can also be reversed by preincubation with vitamin E. While the per-oxidation extent is similar for fibroblasts cultur-ed in monolayers or in dermal equivalent, the cytotoxic response to UVA is more pr'oriouiiced in dermal equivalent.



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